Hello!
I'm Rafael Messina do Rosario, and here I will show you my experience obtained thus far in the translation area.
These are all the projects in which I have participated and am still participating on the Open Source Platform Crowdin
One Piece Pirate Warriors 3 - A game
ExpidusOS File Manager - A file manager
Fatal Frame: Mask of the Lunar Eclipse - A game
Open Food Facts - An application with the objective of cataloguing food products
Eco by Strange Loop Games - A game
Here is a medical excerpt issued by a translation site as a test
A via comum das doenças hepáticas é a cirrose, que pode descompensar e levar ao desenvolvimento de complicações tais como a encefalopatia, que tem sua etiopatogenia relacionada à hiperamonemia desenvolvida pela disfunção hepática. Sugere-se que substâncias capazes de interferir nos níveis séricos de amônia possam ser utilizadas no seu manejo. A cafeína (CFA) aumenta a atividade do ciclo da ureia no fígado, aumentando a degradação de amônia. Além disso, ao antagonizar receptores de adenosina, exerce efeito hepatoprotetor impedindo a formação de fibrose e efeitos de estimulação do sistema nervoso central. A partir disso, o que se propõe com o presente estudo é a análise dos efeitos da CFA sobre a evolução da cirrose biliar secundária induzida por ligadura de ducto biliar comum, correlacionando a sua utilização com o desenvolvimento da encefalopatia causada pela hiperamonemia produzida pela doença hepática. Modelo experimental de cirrose biliar secundária, por meio da ligadura de ducto biliar comum, e de encefalopatia hepática, por meio da administração de ração hiperproteica. Ratos machos da linhagem Wistar (32) foram divididos igualmente em 4 grupos. O experimento durou 28 dias, com administração de CFA 50mg/Kg/dia. Foram realizados exames laboratoriais, análise histológica de fígado e encéfalo, teste comportamental de campo aberto (TCA) e análise comportamental diária.
And now the translation of the medical excerpt issued by the translation site
The common pathway of liver diseases is cirrhosis, which can decompensate and lead to the development of complications such as encephalopathy, which is etiopathogenically related to hyperammonemia developed by hepatic dysfunction. It is suggested that substances capable of interfering with serum ammonia levels can be used in its management. Caffeine (CFA) increases urea cycle activity in the liver, promoting ammonia degradation. Additionally, by antagonizing adenosine receptors, it exerts a hepatoprotective effect, preventing fibrosis formation and central nervous system stimulation effects. Based on this, the purpose of this study is to analyze the effects of CFA on the progression of secondary biliary cirrhosis induced by common bile duct ligation, correlating its use with the development of encephalopathy caused by hyperammonemia produced by the hepatic disease. An experimental model of secondary biliary cirrhosis was induced by common bile duct ligation, and hepatic encephalopathy was induced by the administration of a hyperproteic diet. Male Wistar rats (32) were equally divided into 4 groups. The experiment lasted 28 days, with the administration of CFA at 50mg/Kg/day. Laboratory tests, histological analysis of liver and brain, open field behavioral test (OFT), and daily behavioral analysis were performed.
